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Hemostatic medicine, has also angioprotective and proagregantic action. Stimulates the formation of platelets and their exit from the bone marrow. Haemostatic effect due to the activation of thromboplastin formation at the site of damage to small blood vessels and decrese in the vascular endothelium of prostacyclin PgI2, promotes adhesion and aggregation of platelets, which ultimately leads to a stop or reduce of bleeding. Increases the rate of formation of the primary thrombus and increases its retraction. Does not affect the concentration of fibrinogen and prothrombin.
